1992-1996：华中农业大学，土壤与植物营养专业，学士
1996-2002：华中农业大学，微生物学专业，博士
2002-2005：法国里昂第一大学感染与免疫学系，博士后
2005-2009：美国克利夫兰临床医院，分子遗传学系，博士后
2009-至今：武汉大学生命科学学院，病毒学国家重点实验室，教授

1. 国家杰出青年科学基金项目（医学部）（基金号：81825015）2019-2023（主持），400万
2. 国家自然科学基金重点项目（基金号：31630086）2017-2021（主持），345万
3. 国家自然科学基金面上项目（基金号：81471939）2015-2018（主持），100万
4. 国家自然科学基金面上项目（基金号：81271816）2013-2016（主持），70万
5. 国家重点研发计划重点专项（基金号：2017YFA0505800）2017-2022（骨干），130万
6. 国家科技重大专项“艾滋病和病毒性肝炎等重大传染病防治”（基金号：2018ZX10101004） 2018-2020 （骨干），114万
7. 教育部新世纪优秀人才支持计划 2014-2016（主持），50万
8. 湖北省自然科学基金创新群体（基金号：2017CFA022）2017-2019（主持），50万
9. 湖北省杰出青年基金 2014-2016（主持），10万

1. Yang X, Hu Z, Zhang Q, Fan S, Zhong Y, Guo D, Qin Y, Chen M*. 2019. SG formation relies on eIF4GI-G3BP interaction which is targeted by picornavirus stress antagonists. Cell Discov. eCollection.

2. Zhang L, Qin Y, Chen M*. Viral strategies for triggering and manipulating mitophagy. Autophagy. 2018. 14(10):1665-1673.

3. Zhang S#, Cheng Q#, Luo C, Qin Y*, Chen M*. Human Parainfluenza Virus Type 3 Matrix Protein Reduces Viral RNA Synthesis of HPIV3 by Regulating Inclusion Body Formation. Viruses. 2018. 11;10(3).

4. Hu Z, Wang Y, Tang Q, Yang X, Qin Y, Chen M*. Inclusion bodies of human parainfluenza virus type 3 inhibit antiviral stress granule formation by shielding viral RNAs. PLoS Pathog. 2018. 14(3): e1006948.

5. Yang X, Hu Z, Fan S, Zhang Q, Zhong Y, Guo D, Qin Y, Chen M*. Picornavirus 2A protease regulates stress granule formation to facilitate viral translation. PLoS Pathog. 2018. 14(2): e1006901.

6. Zhang S#, Cheng Q#, Luo C, Yin L, Qin Y*, Chen M*. An alanine residue in human parainfluenza virus type 3 phosphoprotein is critical for restricting excessive N0-P interaction and maintaining N solubility. Virology. 2018. 518:64-76.

7. Ding B#, Zhang L#, Li Z, Zhong Y, Tang Q, Qin Y, Chen M*. The Matrix Protein of Human Parainfluenza Virus Type 3 Induces Mitophagy that Suppresses Interferon Responses. Cell Host & Microbe. 2017. 21(4):538-547.

8. Zhang S#, Jiang Y#, Cheng Q, Zhong Y, Qin Y, Chen M*. Inclusion body fusion of human parainfluenza virus type 3 regulated by acetylated α-tubulin enhances viral replication. Journal of Virology. 2017. 91(3). pii: e01802-16.

9. Jiang Y, Qin Y*, Chen M*. Host-Pathogen Interactions in Measles Virus Replication and Anti-Viral Immunity. Viruses. 2016. 8 (11), E308 (Invited review).

10. Yan Q#, Wu L#, Chen L, Qin Y*, Pan Z*, Chen M*. Vesicular stomatitis virus-based vaccines expressing EV71 virus-like particles elicit strong immune responses and protect newborn mice from lethal challenges. Vaccine. 2016. 34:4196-4204.

11. Zhang G#, Zhong Y#, Qin Y, Chen M*. Interaction of Human Parainfluenza Virus Type 3 Nucleoprotein with Matrix Protein Mediates Internal Viral Protein Assembly. Journal of Virology. 2015. 90(5):2306-15.

12. Ding B, Qin Y, Chen M*. Nucleocapsid proteins: roles beyond viral RNA packaging. WIREs RNA. 2016. 7(2):213-26. (Invited review).

13. Chen L, Zhong Y, Hu Z, Qin Y. Chen M. Chen M*. Two second-site mutations compensate the engineered mutation of R7A in vesicular stomatitis virus nucleocapsid protein. Virus Research. 2016. 214:59-64.

14. Chen L#, Yan Q#, Lu G, Hu Z, Zhang G, Zhang S, Ding B, Jiang Y, Zhong Y, Gong P, Chen M*. Several residues within the N-terminal arm of vesicular stomatitis virus nucleoprotein play a critical role in protecting viral RNA from nuclease digestion. Virology. 2015. 478:9-17.

15. Ding B, Zhang G, Yang X, Zhang S, Chen L, Yan Q, Xu M, Banerjee AK, Chen M*. Phosphoprotein of human parainfluenza virus type 3 blocks autophagosome-lysosome fusion to increase virus production. Cell Host & Microbe. 2014. 15(5):564-77.

16. Zhang G, Zhang S, Ding B, Yang X, Chen L, Yan Q, Jiang Y, Zhong Y, Chen M*. A Leucine Residue in C-terminus of Human Parainfluenza Virus Type 3 Matrix Protein Is Essential for Efficient Virus-Like Particle and Virion Release. Journal of Virology. 2014. 88(22):13173-88.

17. Zhang S, Chen L, Zhang G, Yan Q, Yang X, Ding B, Tang Q, Sun S, Hu Z, Chen M*. An amino acid of human parainfluenza virus type 3 nucleoprotein is critical for template function and cytoplasmic inclusion body formation. Journal of Virology. 2013. 87(22):12457-70.

18. Chen L, Zhang S, Banerjee AK and Chen M*. N-Terminal Phosphorylation of Phosphoprotein of Vesicular Stomatitis Virus Is Required for Preventing Nucleoprotein from Binding to Cellular RNAs and for Functional Template Formation. Journal of Virology. 2013. 87(6):3177-86.